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Leading researchers are specially invited to provide a complete understanding of the key topics in these archetypal multidisciplinary fields. In a form immediately useful to scientists, this periodical aims to filter, highlight and review the latest developments in these rapidly advancing fields.
Leading researchers are specially invited to provide a complete understanding of the key topics in these archetypal multidisciplinary fields. In a form immediately useful to scientists, this periodical aims to filter, highlight and review the latest developments in these rapidly advancing fields.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
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