A Meta-analysis about EEG-Alpha-Asymmetries and Depression in Adults
Proof of a vulnerability marker for depression?
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Research about EEG-Asymmetries in infants,adolescents and adults searched from the beginningfor a vulnerability marker of depression.EEG-Alpha-Asymmetries in the frontal anterior regionsof the brain are a potential candidate for such avulnerability marker. But research results presentedtill today are heterogenous and not consistent intheir findings. The present work tried to bringevidence of a state-independent vulnerability markerfor depression reflected in activation asymmetries.Studies examining electroencephalographic (EEG) alphaactivity in subclinical, clinical and remitteddepressives wer...
Research about EEG-Asymmetries in infants,
adolescents and adults searched from the beginning
for a vulnerability marker of depression.
EEG-Alpha-Asymmetries in the frontal anterior regions
of the brain are a potential candidate for such a
vulnerability marker. But research results presented
till today are heterogenous and not consistent in
their findings. The present work tried to bring
evidence of a state-independent vulnerability marker
for depression reflected in activation asymmetries.
Studies examining electroencephalographic (EEG) alpha
activity in subclinical, clinical and remitted
depressives were analyzed in order to see whether
effect sizes can be found for the relationship
between activation asymmetries and depression and of
what magnitude they are.
adolescents and adults searched from the beginning
for a vulnerability marker of depression.
EEG-Alpha-Asymmetries in the frontal anterior regions
of the brain are a potential candidate for such a
vulnerability marker. But research results presented
till today are heterogenous and not consistent in
their findings. The present work tried to bring
evidence of a state-independent vulnerability marker
for depression reflected in activation asymmetries.
Studies examining electroencephalographic (EEG) alpha
activity in subclinical, clinical and remitted
depressives were analyzed in order to see whether
effect sizes can be found for the relationship
between activation asymmetries and depression and of
what magnitude they are.
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